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Mammalian epidermis is maintained through proliferation of stem cells and differentiation of their progeny.
Alternatively, MBP turn-over rates may be faster and MBP levels actively maintained through proliferation and differentiation of the oligodendrocyte precursor cell compartment.
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Because pTyr142 was maintained through mitotic proliferation after the depletion of BAZ1B in embryonic germ cells, these results suggest that, contrary to our prediction, BAZ1B is not required for Tyr142 phosphorylation in male germ cells.
Evidence is accumulating that the reservoirs may consist of truly latent provirus maintained in long-lived quiescent CD4 memory T cells, seeded soon after acute infection or maintained through homeostatic proliferation (Maldarelli et al., 2014; Wagner et al., 2014; Cohn et al., 2015).
The prevailing view that blood monocytes give rise to tissue macrophages in the steady state has been challenged by recent evidence in the mouse that phagocytic cells arising in the yolk sac can populate tissues during development and can be maintained through local proliferation [ 2, 3].
Since MYC induction in MB cells leads to an increased proportion of cells undergoing programmed cell death, while overall viability is maintained through higher rate of proliferation [ 20], our results suggested that JAG2 might be one of the proteins involved in the regulation of MYC-controlled apoptosis or proliferation.
Epidermal homeostasis and barrier integrity is maintained through the coordinated regulation of proliferation, migration and cell death.
Tissue homeostasis is maintained through a balance between cell proliferation and apoptosis.
The human oesophageal stratified squamous epithelium is maintained through an exquisite balance between proliferation and terminal differentiation. 1 Most of the current knowledge on tissue homeostasis and injury repair is based on murine models; however, there are fundamental differences between mouse and human oesophagus.
Moreover, cell proliferation can be maintained through the transient modulation of downstream signaling.
Expression of uromodulin was maintained through the first 24 hours when proliferation is known to begin [ 16].
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