Sentence examples for maintained mitochondrial function from inspiring English sources

In this model, the neuroprotective capacity of Cr in the hypoxic perinatal brain is attributed to reduced lipid peroxidation and apoptosis, likely through maintained mitochondrial function [71].

Wang et al. (2008) have shown that RIC in vivo using limb preconditioning generated a dialysate, which protected naïve perfused rabbit hearts against the myocardial IRI in terms of preserved outer mitochondrial membrane (OMM) integrity and maintained mitochondrial function.

In addition, inorganic phosphate increased caspase-3 activity and decreased the mitochondrial membrane potential, whereas NOS inhibitors maintained mitochondrial function, illustrating that NO mediates phosphate-dependent chondrocyte apoptosis [ 29].

Taken together, these data identify Clu as a mitochondrially-associated protein that plays a direct role in maintaining mitochondrial function and that binds TOM20, and support a role for Clu linking mitochondrial function to the PINK1-Park pathway.

We also demonstrate that Blm10 acts to maintain mitochondrial function.

Chen, Y.-C. Y.-C.. Msp1/ATAD1 maintains mitochondrial function by facilitating thetdegralation of Msp1/ATAD1ed tail-anchored proteins.

These processes serve to maintain mitochondrial function and ensure optimal energy supply for the cell.

These data demonstrate that tumour elicited c-Kit signalling drives both the increase in neutrophil number and mitochondrial fitness resulting in an oxidative population of neutrophils which use fatty acid metabolism to maintain mitochondrial function.

Conclusion: The results indicate that preterm fetuses are more capable than term fetuses of maintaining mitochondrial function under conditions of transient intrauterine ischemia and suggest that oxygenderived free radicals may play a crucial role in the development of neonatal neurologic deficit.

We examined the critical roles played by the antioxidant, non-protein thiol, glutathione and related enzyme, glutaredoxin in maintaining mitochondrial function during excitotoxicity caused by β-N-oxalyl amino-l-alanine (l-BOAA), the causative factor of neurolathyrism, a motor neuron disease involving the pyramidal system.

In other terms, the failure of maintaining mitochondrial function through biogenesis may contribute to bad outcome.