Sentence examples for maintain low expression from inspiring English sources

In the absence of TGFβ, the cells maintain low expression levels of pro-fibrogenic genes (Fig.  3c).

We find that the cells grown in mRM maintain low expression levels of Acta2, Col1a1, and Lox, similar to uncultured D0 mouse qHSCs and have an intermediary phenotype with large intra-cytoplasmic lipid droplets (Additional file 1: Figure S6).

Incubation with proteasomal inhibitor N-[N- N-Acetyl-L-leucyl -L-leucyl]-L-norleucine (ALLN-[N- N-Acetyl-L-leucyl -L-leucyl]-L-norleucine, suggestiN-[N- N-Acetyl-L-leucyl -L-leucyl]-L-norleucine level of HMGR by proteasomal degradatioN-[N- N-Acetyl-L-leucyl -L-leucyl]-L-norleucine

From 0 to 2 dpi, MbIGYP11, 13, 16, 20, 39, 73, and 76 maintained low expression levels without significant induction.

In contrast, Cla005425 maintained low expression throughout fruit development and ripening, decreasing further in the later stages.

Lipoprotein lipase (Lpl) and Fabp3 gene expression were dramatically decreased 3 days after SCI as compared to controls and maintained low expression levels on days 8 and 14 after SCI.

Figure 5A shows that the expression level of CcGCC1 in clementine increased about 15-fold during flavedo ripening, while 39B3 mutant maintained low expression levels in November and only experienced a slight increase in January.

Unlike miR160, miR167 and miR390 maintained low expression levels from SD8 to C5, and then increased dramatically from C5 to LD3, indicating that miR167 and miR390 may function in the auxin pathway during active growth.

Cluster 2 comprises 337 genes that were suppressed immediately at stage 2 and most maintained low expression levels at stage 3. BLAST analysis indicated that these genes are involved in amino acid metabolism, development and transcription, and auxin signaling.

Functional Shh signalling was confirmed, as the hedgehog target, Gli-1, was re-expressed in the base and stroma of pseudopyloric metaplastic lesions in H. felis-infected mice, in contrast to Gli-3, which is not a transcriptional target of the Shh pathway, and which maintained low expression.

In the absence of IL-12 receptor signaling, we show that KLRG1+ CD8+ T cells maintain low CXCR3 expression.