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This underlines the importance of the ability of a resuscitation fluid to maintain capillary perfusion.
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PEG-albumin maintained capillary perfusion during the initial stages of hemorrhage and was able to re-establish functional capillaries at the end of hemorrhage.
Therapeutic means of maintaining capillary perfusion, both prior to hospitalization and in relation to recanalization, could therefore prove to be important in the management of acute cardiac syndrome.
These alterations are characterized by a decrease in capillary density and in heterogeneity of capillary perfusion with stopped-flow capillaries in close vicinity to well-perfused capillaries.
Capillary perfusion rate (CPR; 1/1) was determined as the length of continuously perfused microvessels per total length of capillaries in the observational area.
Causes of reduced capillary perfusion were therefore explored.
Supra-physiological oxygen tensions also alter the microperfusion through a decreased capillary perfusion [11] and the systemic perfusion.
This is important in arterioles, since arteriolar tone controls capillary perfusion.
Hydrocortisone may induce some degree of arteriolar vasoconstriction and this could alter capillary perfusion.
Oversupply of O2 to arteriolar walls may cause constriction and paradoxically reduced capillary perfusion.
Leakage of dye in posterior uveitis can limit our ability to evaluate adjacent capillary perfusion and can be present due to damaged or ischemic capillary beds.
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