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Three target regions were identified in the main analyses (see Figure 2) as exhibiting differences in BLA connectivity between posttraumatic stress disorder (PTSD) and trauma-exposed control groups.
We therefore performed the main analyses (see below) separately for each genus Four coding regions (rpoB, rpoC1, rbcL and matK) required the use of multiple primer sets for amplification.
In addition to the variables included in the main analyses (see above), we also included cumulative NNRTI use in the analysis of the effects of PI on APRI, and similarly included cumulative PI use when analyzing the effects of NNRTI on APRI.
Variations between subgroups, however, remained similar to the main analyses (see online supplementary table S1).
Despite a considerable reduction in the number of cases in these analyses, the estimates were largely similar to the main analyses (see supplementary table 5).
However, results of sensitivity analyses that excluded rice consumers were generally consistent with the main analyses (see Supplemental Material, Table S3).
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For behavioral and main effect analyses see Supplementary Data.
For behavioral and main effect fMRI analyses see Supplementary Data.
First, we repeated the analysis of the 'main composite' in population 3 and compared the findings with the main analyses to see to what extent progressively restricting the populations had an effect on the estimated relative risks.
For an overview of the data production process and the main types of possible analyses see, e.g. Rey (2016).
The fixed model (Table 7) considered how RR estimates varied by six main characteristics and additional analyses (see Additional file 5: Detailed Analysis Tables) tested whether adding in further characteristics improved the model fit.
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