Excess extracellular volume is a major clinical problem in patients with chronic kidney disease (CKD) and causes lower extremity edema, hypertension, pulmonary vascular congestion or edema, and heart failure [ 1].
Two patients had painless lower limb edema and 4 had unilateral lower limb edema with pain (grade 3 in 3 patients and grade 4 in 1 patient) prior to intraarterial chemotherapy.
Two noteworthy side effects are lower extremity edema in patients treated with endothelin receptor antagonists [ 5] and pulmonary edema in patients with pulmonary veno-occlusive disease treated with parenteral prostacyclin analogs [ 6].
The analbuminemic trait was diagnosed in a young Turkish woman on the basis of her clinical symptoms (bilateral lower limb edema) and biochemical findings (minimal albumin amount and variable increases in other protein fractions).
Chronic lower extremity edema is one of the most common medical problems; every second person has the problem, and every eighth has advanced problems that require treatment.1 Treatment interventions for wounded patients and prevention plans to prevent recurrence abound in the literature.
A right inguinal bulge was noted, and there was no lower limb edema.
The patient denied chest pain, lower extremity edema, weight loss, or episodes of syncope.
External exam demonstrated significant upper and lower lid edema and erythema, ptosis, and moderate proptosis.
Stable hemodynamically, tricuspid focus regurgitation murmur, lower limb edema, tension ascites and bilateral pleural effusion.
There was trace lower extremity edema at the right pretibial region.
Postoperatively, mild lower limb edema was observed without signs of phlegmasia that improved with compression stockings.
