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Expression of both c-Fos and MHCI-HC was uniform, albeit at a very low level in control animals.
Conversely, BID, which was expressed at low level in control cells, showed a significant increase in treated cells.
Cox8b was expressed at a very low level in control iWA, but NE/Rosi treatment led to a very substantial increase in expression (Fig. 4 C ).
The brown/ brite -specific mitochondrial protein UCP1 was expressed at a low level in control iWA, but upon chronic NE/ brite -specificUCP1 was strongly (∼700-fold) induced (Fig. 4 E ; note the logarithmitochondrial
A strong increased transcript abundance of the phosphate transporter MtPt4 and the phosphate-responsive ubiquitin-conjugating enzyme PHO2 was detected for arbuscule and the adjacent non-colonized cells, but only at a very low level in control cells of non-mycorrhizal roots [ 19].
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In particular, two genes that may function in tumorigenesis, sox-2 and midkine, were expressed at low levels in control cells but upon 5-aza-2-deoxycytidine treatment these genes became overexpressed.
Interestingly, Fzd2 and 3 were expressed at low levels in control as well as IPF lungs, but significantly increased in IPF (log-fold change 1.04±0.24 and 1.41±0.31, respectively) (Figure 1b).
PpHsp16.4 was found to be constitutively expressed at relatively low levels in control gametophytes.
Aβ (Fig. 6A,K), APOJ (Fig. 6D,N), and APOE (Fig. 6E,O) were expressed at relatively low levels in control conditions.
The female-specific tra F splice form can be detected at low levels in control males by qPCR, at a few percent of the level found in females.
Although these proteins were present at very low levels in control endothelial cells, increased signal was detected in Ezh2-deficient endothelial cells (Fig. 3C).
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