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However, a small amount of low frequency power loss was noticed even when tests were repeated with low intensity sound.
Furthermore for low intensity sound, phase locking of synaptic vesicle release seems to happen at the subharmonics of the fundamental.
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AOAs were larger in blocks with low intensity sounds than in blocks with high intensity sounds.
Low-intensity sound can get past your ear the same way that high-speed light can get past your eye.
To examine this issue, we had volunteers indicate the presence or absence of a brief, low-intensity sound that was either presented alone or paired with a simultaneous light in a one-interval signal detection task.
Moreover, shorter response durations to low-intensity sounds made the 'two events' practically overlying.
In the two bimodal conditions low-intensity sounds also evoked greater AOAs than high-intensity sounds (F 1,8) = 8.73, p<0.02), with a similar right-hemisphere bias (F 1,8) = 6.73, p<0.05).
AOAs occurred reliably in auditory attention conditions and were enhanced during attention to unimodal auditory sequences and during the more difficult auditory-attention conditions with low-intensity sounds.
The only stimulus parameter that reliably modulated AOA ROI activity was sound intensity: right hemisphere AOAs were larger during the more difficult auditory tasks with low-intensity sounds (F 1,8) = 14.60, p<0.01).
Thus, one explanation of AOA enhancements to low-intensity sounds is that they reflected the increased engagement of sustained auditory attention during the more difficult low-intensity task conditions.
In such situation two leaves would represent severe obstacle to any low intensity air-borne sounds.
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