Sentence examples for loss of axis from inspiring English sources

By contrast, loss of cohesin causes a very specific loss of axis structure and decreased DSB formation.

The abnormalities in FMS patients are consistent with loss of HPA axis resiliency.

Potential participants were excluded for any contraindications to magnetic resonance imaging (MRI) (e.g., pacemaker), history of head injury with more than 10 minutes loss of consciousness, Axis I psychiatric disorders (e.g. schizophrenia, bipolar disorder), substance abuse and/or dependence, and renal failure.

In conclusion, our results demonstrate that accumulation of WT p53 leads to repression of Toca-1, and loss of this regulatory axis likely contributes to upregulation of Toca-1 in highly metastatic cancers.

In addition, it has been reported that miR-205-5p miR-205-5p miR-205-5pa p53 famisy memberegulatedng in epithelial to mesenchymal transition inhibytion, whereas the loss of the p63/miR-205 axis enhances cell migration and metastasis in p53state cancer cells.

Strikingly, despite the loss of transcriptional focusing of axis association sites in the absence of cohesin and the overall altered patterns of Red1 distribution, DSB formation within Red1 clusters occurred at the same hotspots as in wild-type cells.

The neuropeptide Kp and its cognate receptor, GPR54 are major regulators of the HPG axis and loss-of-function mutations in GPR54 were found to cause an absence of puberty and hypogonadotropic hypogonadism [2], [3].

Kp and GPR54 are established regulators of the hypothalamic-pituitary-gonadal (HPG) axis and loss-of-function mutations in GPR54 are associated with an absence of puberty and hypogonadotropic hypogonadism, thus defining an important role of the Kp/GPR54 signaling system in reproductive function.

To understand how the differences in miR-9 loss-of-function phenotype along the AP axis arise at molecular level, we set to determine the potential miR-9 targets in X. tropicalis in relation to the phenotype we observed.

In germline stem cell (GSC) in the ovarioles and testis, the contact of stem cells with the tightly regulate stem cell number and coordinated maturation of the progeny to form gametes. Loss of regulation in this axis results in mis-regulated cell number amplification (López-Onieva et al., 2008; Wang et al., 2008; Losick et al., 2011).

Without loss of generality, the y-axis is taken perpendicular to the plates, whereas the planes (y=-L) and (y=L) represent the lower and upper plates, respectively (see Figure 1).