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Residual long-term abnormalities following retirement required investigation.
13 However, the residual and long-term abnormalities following retirement are unknown.
Our results demonstrated residual long-term abnormalities following an average of 15 years' retirement, with a time-related exposure response.
This detailed follow-up study conducted 5 years after initial presentation was designed to investigate possible characteristics underlying the aetiology of rickets and long-term abnormalities in bone and mineral metabolism.
Although VCM exposure was already known to affect microcirculation, our study demonstrates residual long-term abnormalities following an average of 15 years' retirement, with a time-related exposure response.
Although VCM exposure was already known to affect microcirculation, our study demonstrated the potential for residual long-term abnormalities following an average of 15 years' retirement, with a time exposure response.
Because of this, it is difficult to evaluate whether the short- and long-term abnormalities (e.g. post partum haemorrhage and chronic abdominal pain) were directly due to the caesarean sections or to underlying conditions.
Both of these definitions however, predict long term respiratory abnormalities [ 24].
More recently, the use of oxygen dependency at 36 weeks postconceptional age (PCA) has been proposed as a more suitable definition of BPD.[ 27] Both definitions of BPD predict long term respiratory abnormalities.
In the long term, this abnormality is known to depress cardiac function and induce severe cardiomyopathy [ 5].
The present data demonstrate that damage to MD in the first postnatal week results in long term behavioral and cognitive abnormalities in adult rats, presumably by disrupting circuitry forming between the MD and PFC during this critical period in brain maturation [25,38].
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