Sentence examples for livers with increased from inspiring English sources

Coupling the preexisting steatosis in STAT5-deficient livers with increased adipose tissue-derived lipid fluxes causes the spontaneous development of liver tumors in DKO mice.

Imaging findings indicative of cirrhosis included small nodular and irregular livers with increased echogenicity and/or a significant reduction in Doppler flow in the portal circulation on ultrasound, CT, or MRI.

Surgeons can expect to encounter steatotic livers with increased frequency as this reflects the corresponding global "metabolic syndrome" epidemic [ 10, 11].

Posttreatment MRI and hepatobiliary scintigraphy show ascites and shrinkage of the cirrhotic liver with increased arterial enhancement of the treated lobe and decreased 99mTc-mebrofenin uptake (e, f).

The consensus statement described uncommon toxicity in the liver, with increased risk in patients re-irradiated and/or with prior liver disease.

WDHCC was diagnosed mainly based on the following major histologic features proposed by ICGHN and WHO: (1) increased cell density, more than twice that of the surrounding liver, with increased N C ratio; (2) irregular thin-trabecular pattern of growth; (3) pseudoglandular structures; (4) fatty change; (5) unpaired arteries; (6) intratumoral portal tracts; and (7) stromal invasion [ 20, 21].

25– 27 The increased expression of 11β-HSD1 in liver along with increased plasma glucose levels in db/db mice is consistent with the role of 11β-HSD1 in providing an indirect source of glucose.

The diagnosis of liver cirrhosis was made if a small and/or nodular liver along with increased echogenicity and an irregular appearing was found.

More recently, Pcsk9 mice [ 10] were found to be protected against liver metastases, with increased apoptosis of metastatic cells and have a blunted response to liver regeneration following hepatectomy [ 11].

WrnΔ hel /Δ hel mice exhibit increased GSH levels in liver tissue with increased ROS, DNA damage and decrease ATP levels with age.

Advanced NAFLD can eventually progress to end-stage liver disease with increased risk of hepatocellular carcinoma (HCC) (Kopec and Burns, 2011).