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At the same time, apparent ACAT activity levels decreased in foetal and regenerating rat livers when compared with respective control tissues.
Thus, in the TUDCA and PBA groups, and particularly in the TUDCA group, pP38 levels increased in both livers when compared with the PH+I/R group.
PH+I/R increased XBP-1(S) protein levels in steatotic and non-steatotic livers when compared with the sham group.
The studies focused on the comparison of atherosclerotic lesion between IL-1Ra+/+/ApoE−/− and IL-1Ra+/−/ApoE−/−-mice, because IL-1Ra−/−/ApoE−/−-mice were significantly leaner, had significantly elevated total plasma cholesterol, decreased HDL-cholesterol levels, and severe fatty livers when compared with the other mice.
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The migration was more largely observed in coculture with liver when compared to kidney kidney cocultures.
At conventional B-mode ultrasound, diffuse fatty infiltration results in increased echogenicity of the liver when compared to other organs such as the renal cortex (Fig. 1).
NEM treatment alone caused a significant reduction of the liver glutathione levels in group 2. Furthermore, NEM treatment caused congestion and mononuclear cell infiltration in the liver when compared to the control group.
Furthermore, relatively low luciferase expression of GS-PEG-Apt/pGL3 and GS-PEG/HA2-Apt/pGL3 NPs was recorded in the brain, heart, and liver when compared with that of GS-PEG/pGL3, whereas the spleen and kidney levels did not markedly changed.
PLGA CS PEG nanoparticles showed dramatic prolongation in blood circulation, as well as reduced macrophage uptake, with only a small amount of the nanoparticles sequestered in the liver, when compared to PLGA CS and PLGA nanoparticles.
Pre-treatment of animals with tea seed oil (150 g/kg diet) could increase the activities of glutathione peroxidase, glutathione reductase and glutathione S transferase in liver when compared with CCl4-treated group (p < 0.05).
The liposomal formulation was administered by i.p. route at 3 mg/kg for eight days and reduced the parasite burden to 54% in liver when compared to untreated group; no improvement was observed in the spleen of infected hamsters.
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