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Four days post-injection, animals were sacrificed, and the livers were studied for GFP expression and ERα mRNA level.
PND 15 livers were studied as animals of this age have circulating T4 levels higher than at any other age [ 20] and the liver is widely studied as a model of TH action.
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The histone modifications in fatty liver were studied with the activity of HDAC and DNMT.
As the liver consumes more oxygen at rest than other organs and is thought to play a role in the systemic control of glucose and lipid utilization [ 23], gene expression and metabolic measurements in the liver were studied.
Finally, gene expression in liver was studied due to the importance of the organ in overall energy metabolism.
To examine this, we exposed emphysematous rats (the pre-existing emphysema was formed through smoke exposure for 16 weeks) to hypoxia during sleep in a custom-made chamber therefore developing a novo rat model of SH in emphysema (SHE); with this model the inflammatory cytokines of the liver was studied and the four important coagulant/anticoagulant factors just mentioned above were measured.
Initial tumour cell arrest in the liver was studied in more detail by in vivo microscopy.
Here the expression in the liver is studied, as the liver is the major organ in the production of apoliprotein.
Liver was studied because of its significant metabolic relevance and muscle was studied because it is the major store for vitamin B6.
The rate of transformation of catalase to catalase-formate in liver was studied by freeze-clamping liver in anaesthetised chickens, then warming to 37 degrees for 1 or 2 minutes anaerobiosis, and then refreezing.
To investigate whether cavins are involved in hepatic sinusoidal angiogenesis and remodeling during progression to cirrhosis, normal control liver specimens and early and late cirrhotic liver specimens were studied.
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