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Due to high rates of fatty acid oxidation, the ketogenic activity of such livers is also pronounced.

The livers were also analyzed for selected T4-specific UGT gene expression and total UGT enzyme activity.

Kidneys, lungs and livers were also normal in these mice (Figure 4A).

Likewise, the number of B1a progenitor in the newborn livers was also similar between WT and mutant mice (Fig. 3B).

Livers were also perfused with acetate, butyrate or octanoate.

Lungs and livers were also harvested and fixed in 10 % neutral buffered formalin for histological analyses.

Significant weight increase of HF livers was also detected (Fig.  2b).

Acute histopathological changes in DOX-treated livers were also reversed by B. serrata extract administration.

In the present analysis, the livers were also found to be the most affected organ (95.08%, 174/183).

Patients with both insulin resistance to glucose and fatty livers were also found to have hearts with impaired energy metabolism.

Rat livers were also sampled immediately, cleaned up in ice cold saline, and snap-frozen at −80°C prior to analysis.