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In contrast, gp130Δhepa female livers displayed normal liver architecture with the presence of some preneoplastic cells.
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In the present study we found that mice harbouring specific deletion of GSK-3α in skeletal muscle or liver displayed normal sensitivity to insulin and normal insulin signaling.
C/EBPα-deficient mice have a severe loss of liver function; in contrast, the C/EBPα-deficient mice expressing C/EBPβ from the Cebpa locus displayed normal liver function [32].
By contrast, livers isolated from mice treated with MSCs displayed normal morphology without nodules, much less infiltration of lymphocytes, markedly reduced granulomas, normal weight, and remarkably reduced Fas ligand expression (Fig. 1C,D; Supporting Figs. S1C, S2A,B).
Euthyroid animals displayed normal histoarchitecture of rat liver.
In the I/R + LSTS group, the hepatocytes and sinusoids displayed normal morphology reflecting well preserved liver parenchyma.
Livers of AnxA1 KO mice on a control diet displayed normal histological appearance apart from the sporadic presence of monocyte infiltration (Fig. 3).
However, whereas a deficit was also observed in the lobules of 20-week-old surviving mice (Fig. 3C), the inner liver structure of Alb-Cre-Fxn L3/L–(S) mice at 20 weeks displayed normal SDH activity (Fig. 3D).
Furthermore, in the livers of rats fed on a SP-diet (SP-control and SP-2 g) the morphology of hepatocytes still displayed normal architecture around the central veins, still arranged in cords.
All displayed normal subsequent development.
shRNAscram-transfected cells displayed normal mitotic structures.
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