This study examines a pretreatment strategy to strengthen the hepatic lineage divergence of mesenchymal stem cells (MSCs).
This has been driven by the advent of molecular systematics and the development of new methods able to integrate multiple sources of evidence into biogeographic inference: lineage divergence times, paleogeographic and paleoclimatic information, or the ecological niche of lineages.
Still other researchers who emphasize the framework of the "general lineage concept of species" contend that lineage divergence or differentiation demarcates the beginning of a new species.
Specific patterns in geography and genealogy provide evidence of processes driving lineage divergence often resulting in speciation.
This analysis is congruent with a multiple lineage divergence pattern from ancestral haplotypes, as expected in a radiation.
Many efforts have been made during the last decade to characterize the molecular mechanisms driving lineage divergence.
Genealogical sorting index values (gsi) indicated that morphotypes within all lakes but Ragnerudsjön showed significant lineage divergence (Table 1).
During mammalian preimplantation development, lineage divergence seems to be controlled by the interplay between asymmetric cell division (once cells are polarized) and positional information.
Furthermore, there is increasing evidence for the presence of compartmentalization of HCV populations within infected patients [42] [47], which would further facilitate lineage divergence within a patient.
We find additional evidence for mouse lemur lineage divergence in the striking degree of concordance between mtDNA clades and patterns of nuclear population genetic structure.
For TEs shared among species, times of lineage divergence place upper bounds on the insertion times: no insertion older than 12 MYA is recognizable.
