LF and LS reviewed and completed the manuscript and provided considerable input with reference to neuraltheapy and musculoskeletal health problems.
LF and JMR reviewed the manuscript.
AMT, LF and UM reviewed and critiqued the manuscript and made substantial intellectual contributions to subsequent drafts.
RKY, RM and LF wrote the manuscript, and PL and MSR reviewed the manuscript.
WP, LF, and CP developed the manuscript, which was reviewed by CS, LA, ZJ, MS, and RW.
Records were reviewed by experienced clinicians in cardiovascular medicine (LA, LF).
LF participated in the interpretation of the data and critically reviewed the manuscript.
Lf resists proteolysis in the infant gastrointestinal tract and uptake of both Lf and bLf has been demonstrated in vitro using an intestinal enterocyte model (reviewed in [ 3]).
To confirm the functional presence of PRLR in MDA-MB-231 cells, we compared protein levels to exogenously introduced PRLR LF expression in CHO-K1 cells, which exhibit low levels of endogenous PRLR (reviewed in [ 10]).
Numerous studies have demonstrated the prognostic power of the high- (HF) and low-frequency (LF) spectral peaks (or their time-domain equivalents [5]) to predict mortality in cardiac patients, especially congestive heart failure (CHF) patients (reviewed in [6], [7]).
