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Levels of TN and TP were nearly twofold higher in the downstream karst reaches.
The plasma levels of TN and anti-PEG IgM antibody were determined by HPLC and ELISA, respectively.
Soil samples showed the lowest levels of TN% compared to plant samples as shown in Fig. 4.
The 08941, 05342, and 04624 mutants all show increased levels of Tn I transcripts.
None of the gene depletions seemed to significantly reduce the basal levels of Tn in HeLa cells.
The lack of a direct correlation indicates that, in human lung and breast tumours, ERK8 levels do not strictly control the levels of Tn.
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Following 16 days of in vitro conditioning with Skov-3 cell medium the MSC express high levels of Tn-C, TSP and FSP and increased their expression of α-SMA, FAP and desmin.
Elevated levels of TN-C have been suggested to induce inflammatory mediators and promote ECM degradation in OA patients [ 39].
Increased levels of TN-C in the joint fluid significantly correlated with cartilage TN-C mRNA and protein levels in OA patients (Table 1).
The levels of TN-C, Bcl-2, cIAP1, and EGFR mRNA expression reached more than 100 times the corresponding levels of the control values in MKN45/PAR1.
Cells that were able to actively spread and form focal adhesions or to generate sufficient tension showed increased levels of TN-C promoter activity as measured using promoter-based reporter constructs, semi-quantitative RT-PCR and microarrays [ 8- 10].
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