Sentence examples for levels of the matrix from inspiring English sources

Measurements are taken at two different indentation depths, which represent different levels of the matrix effects and lead to the establishment of two independent equations that correlate the fiber/matrix properties with the indentation responses.

We have identified key roles for PECAM-1 in both the regulation of HSC migration to the chemokine CXCL12, as well as maintaining levels of the matrix degrading enzyme MMP-9 in the bone marrow vascular niche.

During IL-12 therapy, tumour levels of the matrix metalloprotease MMP-9 declined and its inhibitor TIMP-1 was strongly induced.

Furthermore, ROS have been found to modulate the activity and expression levels of the matrix metalloproteinases in vascular smooth muscle cells that contribute to physiological and pathological vascular remodeling (Fu et al., 2001; Grote et al., 2003).

56 Finally, a study in 161 patients with knee osteoarthritis showed that levels of the matrix metalloproteinases MMP-1 and MMP-3 were predictive of cartilage volume loss as evaluated by quantitative MRI over 2 years (both p<0.05).

Furthermore, the levels of the Matrix metalloproteinase 1 (MMP1) [ 33] were elevated at the basal side of the epithelium showing that, similarly to checkpoint aneuploid mutants, inhibition of apoptosis leads to the activation of MMP1 expression [ 24].

Importantly, the talin2 siRNAs did not affect the levels of the matrix-metalloproteinases MT1-MMP, MMP2 and MMP9 (Fig. 4A).

The macroscopic stress level around a crack-tip is reduced by the debonding damage while the microscopic stress level of the matrix remains unchanged.

This mutation lowers the energy level of the matrix state involved in substrate binding (Fig. 6) and thus the probability of being in that state would be much higher.

We have shown that suppression of Brk expression by RNA interference significantly decreases the secreted level of the matrix degrading enzyme MMP9 and the cytokine VEGFA, suggesting a role for Brk in regulating some of the processes involved in metastasis (proteolytic activity and neo-angiogenesis).

No changes were seen in the expression levels of the cartilage matrix genes, COL2 and AGN, and the OA-associated matrix degeneration genes, MMP2/3/13 and ADAMTS-4/5, under NGF treatment, but treatment with IL-1β significantly increased MMPs (Fig.  4e).