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Levels of cTn were measured at presentation and serially thereafter as long as clinically indicated.
Survivors tended to lower levels of cTN I than nonsurvivors in septic shock, mean 12.4 ng/ml (range 0.5 57.0).
One cannot reiterate often enough the clinical rule that levels of cTn must always be used and interpreted in conjunction with all other clinical information.
Elevated levels of cTn could be due to ongoing myocardial damage or leakage of myofibrillar components and may reflect the loss of viable cardiac myocytes [ 37].
All 10 patients who died in the intensive care unit had elevated levels of cTN I, mean 21.9 ng/ml (range, 2.9 94.1).
We propose that CDC25B and Ctn are targeted to the centrosome together in a multiprotein complex, where the levels of Ctn and other PCM components are regulated at least in part, by CDC25B.
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In patients with ACS (acute coronary syndrome), increased levels of cTns are associated with increased risk of death and re-admissions, and identify patients who benefit from early, intensified treatment [ 1– 3].
At the same time the latencies of messages with a lower priority drop to the next lower level (i.e., CTN 4's latencies drop to the previous level of CTN 3). Figure 9 Exp.
We examined the impact of lowering the level of detection of cTn measurement in consecutive patients presenting to the emergency department (ED) of a large German community hospital.
It is noteworthy that H-DCs produced high levels of various proinflammatory CTN and CHM, while L-DCs were virtually nonproductive.
At study completion, two investigators (IQ, DJC) interpreted ECGs in duplicate, blinded to each others' assessments and to all clinical information with the exception of cTn levels.
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