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The levels of bone turnover markers also changed with age.
Tianeptine, particularly the higher dose, corrected the elevated levels of bone metabolic products and pro-inflammatory cytokines.
Our study correlated bone quality in dialysis patients with levels of bone fluoride, magnesium, and aluminum.
Rats were sacrificed after predetermined periods; serum levels of bone turnover markers were evaluated.
Machined unfilled PEEK had comparable message levels of bone matrix proteins as rough titanium.
The lack of estrogen significantly increased the levels of bone turnover biomarkers and inflammatory mediators.
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Levels of bone-regenerating cells, called osteoblasts, also fell significantly, from 2% before hibernation to just 0.15% during hibernation.
The second aim was to assess the micro-strain induced by different implant geometries and the levels of bone-to-implant contact (BIC) on the surrounding bone.
Moreover, the expression levels of bone-related genes, including type-I collagen, Runx2, osteopontin (OP), and osteonectin (ON), were up-regulated during the osteogenic differentiation of SHEDs.
FGF23 level was independently associated with the levels of bone-metabolism-related markers and vertebral fracture.
There was a correlation between ln FGF23) and the levels of bone-metabolism-related markers (Table 2).
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