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When analysing only patients with septic shock, we found the same correlation between toe-to-room temperature gradient and arterial lactate level, urine output, knee CRT and mottling score.
We collected global hemodynamic parameters (MAP, heart rate (HR) and cardiac index) and microcirculatory dysfunction and organ perfusion parameters (arterial lactate level, urine output, mottling score and knee CRT) at study inclusion and at 6, 12 and 24 h following inclusion.
Serum creatinine level, urine output, and fluid balance were recorded on a daily basis.
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Urine urea nitrogen levels, urine volume, and creatinine levels were measured 24 hours before and after exercise.
Prior to analysis of isoprostane levels urine samples were hydrolyzed and affinity purified.
Notably, most of the patient's values of renal and tubular function (creatinine, urine inorganic phosphate levels, urine calcium: urine creatinine ratio) were within their normal ranges.
The following were measured or performed at the beginning of each year: body mass index; serum total cholesterol, low-density lipoprotein, high-density lipoprotein; triglyceride levels; creatinine (Cr) levels; urine albumin and urine Cr; nonmydriatic fundusgraphy; and semiquantified neuropathy.
Specifically, we observed a statistically significant association between average PM2.5 levels and urine levels of the oxidative damage marker 8-OHdG.
Age, sex, % TBSA burned, plasma NGAL levels, and urine NGAL levels were also independently associated with mortality.
In our study, we used arsenic levels in urine, arsenic levels in drinking water, and skin signs of arsenicosis as exposure indicators.
Urine protein levels: Though urine protein levels at admission were comparable for the two groups (those who became normotensive and those with persistent hypertension) at recruitment, they were higher among those with persistent hypertension.
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