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The outcome suggests that glibenclamide and all the doses of UFG increase the level of SOD, but UFG doses 40 mg/kg was more effective in increase the level of SOD in diabetic rat as compared with different doses of UFG and glibenclamide.
It is possible that the low level of SOD expression in B. neotomae is sufficient to inactivate the host-derived superoxide radicals and any additional increase in the amount of SOD has no functional effect.
On the other hand, Olubodun and Eriyamremu (2015) reported a decreased level of SOD.
In general, the current results show an increased level of SOD activity in water hyacinth plants growing under lead treatment.
The enhancement of H2O2 content may be correlated with the increased level of SOD activity following Pb treatment.
Enhanced level of SOD activity may be attributed to the production of more active oxygen species (AOS) or over expression of genes encoding SOD.
P.tuber-regium administration decreased the level of MDA and increased the level of SOD in the CCl4 treated groups significantly in dose dependent manner.
Just before birth, pancreatic islets exhibited a much lower level of SOD and CAT activity than in liver but, a similar GPX activity.
Our results demonstrated that the level of SOD was increased in the MTX induced rat intestinal mucosal after treated with curcumin (Fig. 6).
However, the level of SOD overexpression in B. neotomae/pBB4BnSOD was clearly lower than that observed in B. neotomae/pBB4SOD (Fig. 6).
Increasing the level of SOD expression in B. neotomae did not alter the bacterial survival in BALB/c and C57BL/6 mice (Fig. 10).
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