Exact(4)
The mechanism explains self-organization of the auxin distribution pattern in an array of functionally identical cells acquiring cell type specialization due to auxin regulation of the level of PIN proteins in these cells, although the orientation of the PIN is assumed already to be established.
Most classification systems of pin site infections are based on clinical signs rather than on microbiological features to grade the level of pin site infection [ 35– 35].
These findings suggest a specification at the level of PIN protein targeting or recycling to retain them at only one membrane after cytokinesis.
DOI: http://dx.doi.org/10.7554/eLife.02860.025 Since auxin had previously been shown to regulate auxin transport at the level of PIN transcription and PIN endocytosis control, we were also interested in examining the role of auxin on PIN phosphorylation.
Similar(56)
ABC's market research apparently showed that keeping questions to the level of flag pins and what your pastor thinks would draw and keep an audience tuned in.
In the present study, 1 mmol/L sodium butyrate inhibited the growth of Phomopsis XP-8 but did not improve the output levels of Pin, PMG, and PDG.
In the bioconversion systems using resting cells, induction with sodium butyrate reduced the output levels of Pin, PMG, and PDG by 0, 32.68, and 23.81%, respectively (Fig. 7c).
In bioconversion systems using the resting cells obtained from the culture treated with ethanol, the output levels of Pin, PMG, and PDG were significantly reduced by 100, 100, and 72.62%, respectively (Fig. 6c).
In the biosynthesis systems containing PDB, induction with sodium butyrate decreased the output levels of Pin, PMG, and PDG by 63.98, 14.61, and 100%, respectively, which correspond to 26.73, 455.12, and 0 μg/L (Fig. 7a).
To determine the changes in auxin polar transport under exogenous auxin treatment, we further analysed the transcript levels of PIN genes.
Although several gene inactivations caused low-to-moderate levels of Pin in one or more of the mutant backgrounds (data not shown), one gene, fbn-1 (ZK783.1), led to strong enhancement of Pin in both non-RNAi-sensitized and RNAi-hypersensitive mutant backgrounds (see below).
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