Sentence examples for lesion ground from inspiring English sources

X-ray diagnostic features of FD are a characteristic hazy bone lesion (ground glass).

The inclusion criteria were age >20, lesion size more than 8 mm, and solid lesions (ground glass opacity (GGO) component of less than 50%).

In our group of pulmonary TB patients, the following associated radiologic characteristics were observed in addition to the PLLs: cavitation (9 lesions), scarring (3 lesions) and ground glass opacities (2 lesions).

Radiographs typically show expansile medullary lesions with ground-glass appearance and areas of sclerosis.

Among radiographic findings, this study revealed that the bronchogenic spread of lesions and ground-glass opacities occupying >25% of total lung volume were associated with miliary TB.

Pure non-solid lesions, or ground-glass opacities, have a low risk of malignancy (10 15%), usually represented by well-differentiated bronchioloalveolar carcinomas (BACs), but their volume and growth are more difficult to evaluate.

Multivariate analysis revealed that age ≤30 years, HIV infection, corticosteroid use, bronchogenic spread of lesions, and ground-glass opacities occupying >25% of total lung volume increased the probability of miliary TB.

To avoid delayed anti-TB treatment, miliary TB should be seriously considered in patients with the factors identified in this study: ≤30 years of age, HIV infection, corticosteroid use, absence of a history of malignancy, bronchogenic spread of lesions, or ground-glass opacities occupying >25% of total lung volume in chest CT scans.

Subsequent multivariate analysis supported the association between miliary TB and age ≤30 years, HIV infection, corticosteroid use, bronchogenic spread of lesions, and ground-glass opacities occupying >25% of total lung volume, as well as the association between miliary nodules of non-miliary TB and a history of malignancy (Table 5).

Our method overcomes the problem of considering actual L/ B and actual lesion volume, being grounded in RC curves determined as functions of PET L/ B m and measured lesion volume, both estimated by an optimized and validated operator-independent technique.

Fabre and colleagues have argued against DSBs as the spontaneous recombination initiating lesion on the grounds that rad52 yku70 double mutants (deficient for both HR and NHEJ) are viable, yet the srs2 sgs1 double mutant is inviable but rescued by loss of HR function, suggesting lethal spontaneous recombination intermediates occur at high frequency (Fabre et al. 2002).