To investigate whether the positive association between adult height and breast cancer is primarily due to an association with leg length, we tested whether the associations with leg length and sitting height were similar in a model including leg length and sitting height as well as covariates (Wald test).
To evaluate whether different common genetic variants influenced both birth and infant length, we tested 2 193 675 million SNPs for association with infant length in almost the same set of samples used for the analysis of birth length (19 studies, N = 28 238; Supplementary Material, Table S7).
For each combination of nucleotide and repeat length, we then tested whether the proportions of expected and observed numbers differed between coding and intergenic regions.
To assess whether the amount of sequence used affected contig length, we performed a test assembly in MIRA with one half of the 454 dataset.
Therefore, using full length YqeH, we tested this possibility in the nucleotide-free, GDP, GTP and GDP·AlFx states.
As we had identified components associated with vesicle trafficking in our co-precipitation experiments with individual domains of Sec7 and the full length protein we tested the secretion of a lysosomal hydrolase and of cAMP phosphodiesterase.
Furthermore, to correct for possible underestimation of the branch length, we generated and tested five additional simulated alignments for each family with all the same parameters except multiplying the branch length by 1.5.
As in the case of full-length TauΔK, we tested different treatment protocols for TauRDΔK mice.
In order to confirm the ability of these antibodies to detect full-length talins, we tested them against lysate from cells expressing either GFP-talin1, GFP-talin2 or GFP alone by Western blotting (Fig. 1D).
To characterize how the accuracy of SDhaP depends upon coverage and haplotype block lengths, we perform tests on simulated data sets.
