Sentence examples for left ventricular load from inspiring English sources

Elevated systolic pressure increases left ventricular load, which results in left ventricular hypertrophy (LVH).

They reduce left ventricular load, suppress the adrenergic stimulus, improve myocardial remodeling, and thereby reduce cardiovascular morbidity and mortality [ 6].

Increased arterial stiffness is an important marker of increased left ventricular load and an independent predictor of cardiovascular morbidity and mortality both in asymptomatic humans (1) and in disease including renal failure (2), hypertension (3), and diabetes (4).

Over time, the increase in stroke volume leads to an increase in left ventricular load, dilation and compensatory left ventricular hypertrophy, a known precursor of heart failure [ 27].

Positive airway pressure settings will also affect right and left ventricular load in both VV-ECMO and VA-ECMO [ 21].

Different deformation modalities such as longitudinal strain [ 14, 15] or torsion [ 16- 18] can be modified by left ventricular load but even if dipyridamole has several systemic effects that can lead to hemodynamic changes [ 19], we show that there are no blood pressure impacts or heart rate variations on LSR.

Stiffening leads to increased left ventricular (LV) load with hypertrophy, decreased capacity for myocardial perfusion, and increased stresses on small arterial vessels, particularly of brain and kidney.

The large negative intrathoracic pressure generated during inspiration increases left ventricular after-load by impairing systolic emptying.

Stiffening also results in more rapid propagation of the arterial pulse pressure wave through the conductance arteries and can be readily assessed by measuring aortic pulse wave velocity (PWV; a marker of regional large artery stiffness) and central augmentation index (AIx; a composite marker of systemic arterial stiffness and left ventricular systolic loading).

To assess left ventricular inotropism, three load-dependent indices of left ventricular contractility (LVESP, LVEDP, and ± dP/dtmax ) were used.

HIV-negative individuals with the metabolic syndrome develop left ventricular systolic and diastolic load independent abnormalities indicating that MC adversely affects left ventricular function [ 21].