Sentence examples for left expression from inspiring English sources

(C ) Experiment 2, 'color-swap' experiment: Mybl2 -specific shRNA M3 coupled with GFP (right) or RFP (left) expression associated with a significant expansion of Mybl2 -downregulated cells (p=0.007 and p=0.001, respectively).

Left: Expression of a GAL4-inducible shRNA transgene against GFP (UAS-GFP-RNAi) will result in gene knockdown by degrading mRNA of fusion protein; Right: GFP fusion proteins can be targeted for ubiquitination-mediated degradation by a modified ubiquitination system called deGradFP (UAS-NSlmbvhhGFP4 ).

While RIP1-Tag2; RIP1-VEGFB mice presented with a similar number of tumors as RIP1-Tag2 mice (Figure 2c, left), expression of the VEGF-B transgene unexpectedly resulted in a significant reduction in total tumor burden by 39% (Figure 2c, right; 59.0±8.2 mm3 vs 35.7±4.2 mm3; p<0.05).

Acid-regulated, PacC dependent (left); expression gated by PacC at both pH 4 and pH 8 (right).

Principal axis 1, which retained 75.6% of the total variability, sorts genes according their fold change in over (on the right) and under (on the left) expression conditions independently of the stress condition applied.

Virus replication is blocked at a late stage of morphogenesis in mammalian cells, leaving expression of late, as well as early, viral genes unimpaired [29].

In the maize leaves, expression of the At4/IPS1-like RNA showed a very strong negative correlation to phosphate concentration of the leaves under all tested stress conditions.

Consequently, we used teashirt-GAL80 (tsh-GAL80; Clyne and Miesenböck, 2008) to suppress thoracic expression, but leaving expression in the descending interneurons.

(middle), Suppressing Gal4-dependent expression in F cells (ppk23-Gal4, UAS-CD8::tdTomato, vGlut-LexA, lexAop-Gal80 ) leaves expression intact in M cells.

(right), Suppressing LexA-dependent expression in F cells (ppk23-LexA, lexAop-CD2 GFP, lexAop-CD2 GFP-Gal80 ) leaves expression intact in M cells.

Subsequently, EBV establishes a lifelong persistent infection of memory B cells, a process accompanied by epigenetic silencing of most viral coding genes, but leaving expression of non-coding small RNAs unaffected (Thorley-Lawson and Allday, 2008; Paschos et al., 2009).