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In mid-March Sarmin hospital was the lead treatment centre in the aftermath of a massive chlorine attack.
Lead treatment increased superoxide dismutase (SOD) activity in both the leaf and root tissues over control, irrespective of the duration of exposure.
In our experimental design, mice in group A were fed 0.01 mg of lead nitrate before infection with 500 larvae of Ancylostoma caninum, mice in group B were infected with larvae without previous lead treatment, and group C received lead nitrate treatment alone.
Similar response to lead treatment was previously noticed in various plants (Piechalak et al. [2002], [2003]; Brunet et al. [2008]).
In the present study, as the lead treatment was reduced, water hyacinth plant growth rates and overall biomass production decreased.
In general, the current results show an increased level of SOD activity in water hyacinth plants growing under lead treatment.
In case of lead treatment in soil, 36 genes are upregulated and 54 genes are downregulated (Additional file 4: Table S4).
Based on the lead toxicity symptoms and physiological growth of the seedlings, the following doses were finally selected and lead treatment was performed for 10 days.
The effect of different concentrations of lead treatment (100 to 1000 mg/L) on photosynthetic pigments is depicted in Figure 2.
It is most likely that lead treatment induced stomatal closure, triggered over the course of the experiment due to the atmospheric carbon fixing activities that were compromised as a consequence (Brunet et al. [2008]).
Lead treatment also increased p-eNOS and iNOS protein expression.
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