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The lead poisoning rate of population aged 0 18 year old indicated lead exposure level.
The corrected sentence is as follows: Two previous studies on ALAD, BLL, and BP one conducted among occupationally exposed workers (Lee et al. 2001) and the other conducted at lower lead exposure level (Smith et al. 1995)—found no association of ALAD polymorphism and BP outcomes.
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The 20th century saw an increase in worldwide lead exposure levels due to the increased widespread use of the metal.
Bone lead measurement is used as an index of cumulative lead exposure levels.
The same covariates except time-since-baseline and its interaction with lead exposure levels were examined.
Major concerns are the cognitive and neurobehavioral deficits resulting from lead exposure levels that were previously considered safe.
The lead workers with at least one minor frequency allele on VDR Bsm1 showed worse renal function with higher lead exposure levels.
These results were found among women with low lead exposure levels that were similar to their same-age peers in NHANES data.
We observed no increase in the odds of GH or preeclampsia or any hypertension related to increasing lead exposure levels (data not shown).
In several countries, lead exposure levels have been in strong decrease over recent years, as documented for instance for Italy (Alimonti et al. 2011).
Differences in the association between urine cadmium and kidney measures by blood lead levels were not evaluated, although lead exposure levels in this occupational population (mean blood lead, 23.1 μg/dL) were substantially higher than in our study.
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