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The latter studies demonstrated that PKA stimulation increased the activity of the 26S proteasome via subunit phosphorylation and/or transcription.
The latter studies demonstrated the nuclear transport of endogenous Smad proteins to the nucleus shortly after TGF-β treatment, even in the absence of Smad4 (as in MDA-MB-468 cells) or in the presence of EGF, activated ras, constitutive raf, or pathologically overexpressed HER-2 (data not shown; Fig. 6).
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The latter studies demonstrate that subunits which are integral to the membrane are important for quinone reduction also, perhaps because they are required to accommodate the isoprenoid chain.
The latter study demonstrated that changes in mRNA stability modulate the stress response together with the TR dynamics for particular gene subsets [4].
Contradicting earlier reports, this latter study demonstrated that the non-essential human disease genes showed no tendency to encode hubs in the human interactome.
This latter study demonstrated no ethnic differences for the incidence of PDN (15).
Furthermore, the latter study demonstrated that clusterin could not distinguish between AD and vascular as well as all-cause dementia.
The latter study demonstrated that AZFc rearrangements were one of the major motifs for large-scale Y structural variation.
This latter study demonstrated a 54% ORR, 7.6 months PFS and 14.7 months OS vs 67.5%, 13 and 18 months, in this study, respectively.
The latter study demonstrated the direct role of NAADP in VEGF-induced Ca2+ mobilization from acidic organelles and its important involvement in the control of angiogenic processes [ 32].
This latter study demonstrated a crosstalk between cardiac fibroblast and cardiomyocytes through extracellular vesicles highly enriched in miR-21*, a documented inducer of cardiac hypertrophy [ 26].
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