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Recently, our laboratory has reported several classes of small molecules that have the ability to inhibit ice recrystallization.
The laboratory has reported small amounts of uranium-238 and plutonium, among other contaminants, in the area of the treatment plant.
Our laboratory has reported that miR-29b participates in aortic root/ascending aorta extracellular matrix remodeling during early aneurysm formation in Fbn1(C1039G/+) Marfan mice.
Prior work from this laboratory has reported anomalous differences in the viscoelastic responses between temperature-jump formed glasses and carbon dioxide pressure-jump or relative humidity formed glasses.
Previously our laboratory has reported on a preclinical post-laminectomy model that mimics the postoperative proliferative fibrotic response grossly, as well as by biochemical assessment of the collagen content within the spinal canal.
Research from this laboratory has reported that extracellular dopamine concentrations in striatum and nucleus accumbens are greater in female rats that are engaging in paced sex compared with those engaging in nonpaced sex.
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Previous studies from our laboratory have reported that exogenous treatment of infected mice with IL-7 augments CD8+ T cell response against T. gondii resulting in their ability to survive lethal infection [6].
Earlier studies in our laboratory have reported the isolation and purification of sperm agglutinating factor (SAF) from E. coli [ 7].
Notably, the Tufro laboratory had reported instead that Sema3C promotes glomerular EC adhesion, migration, and proliferation (Banu et al, 2006).
Several studies, including those from our laboratory, have reported cross-talk between S-palmitoylation and phosphorylation or ubiquitination.
Previous studies performed in our laboratory have reported the use of these markers for the identification of circulating breast cancer cells [ 10, 11].
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