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As recently shown by others (Langerholc et al, 2005), inhibitory activity against cathepsin L was only revealed following incubation with millimolar levels of reducing agents.
For example, the change in SV as predicted by the change in PP in response to the added blood volume of 3.0 L (from 3.5 L to 6.5 L) was only ~60% of the actual change in SV.
One patient, with a body weight of 111 kg and an estimated TBW of 66 L was only treated with 30.4 L due to the limitations of the dialysate chamber.
Three SSRs were mapped close to this locus on LG5: CH03a09, Hi04d02 and CH05f06; however, the frequency of 'Type 1' seedlings bringing the incompatible allele of the SSR CH03a09 (115 bp, denoted as l) was only 48.8%, and hence, this marker might not belong to the region linked to 'Type 1' lethality.
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Similarly, pure PEDOT has weak attenuation to EM wave, and the maximum R L is only −14.5 dB at 7 GHz.
A landmark l is a contributive landmark for S if there exists two routes (bar{R_i}) and (bar{R_j}) from (mathrm{ND}(S)), such that l is only on one route of (bar{R_i}) and (bar{R_j}).
In Fig. 4d and e, one can clearly see that pure GN exhibits poor EM wave absorption properties in the range of 2 4 mm, and the maximum R L is only −8.9 dB at 9.5 GHz.
L is only affected by descriptors with a value S < 1.
osa-miR1861a, g, h, j and o are differentially expressed in both tissues, osa-miR1861b, d, f, i and l are only differentially expressed in leaf and osa-miR1861e, k and m are only differentially expressed in stem.
Since Axis 2 has larger reduction ratio of 8.46 than Axis 3 we will check elongation of Axis 2. Considering L is 40 mm and output pulley rotates (pm) 90°, estimated elongation (L'-L) is only 0.06 mm.
In the integrated model, the differentiation signals such as ceramide and FAS-L were only kept between committed cells and corneocytes to simulate the homeostasis of human epidermis, while the other differentiation rules in the previous model were kept unaltered.
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