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An analysis of the experimental data shows that 220Rn attenuation in a flow through charcoal bed is not exponential with respect to the residence time, L/Ua (L: bed depth; Ua: superficial velocity), but follows a power law behaviour, which can be attributed to the occurrence of large voids due to wall channeling in a flow through bed.
The enhanced expression of L-PGDS in the superficial and middle zones of cartilage could potentially be due to the increased level of the proinflammatory cytokine IL-1β in these zones.
As shown in Figures 2a and 2b, the immunostaining for L-PGDS was located in the superficial and upper intermediate layers of cartilage.
One group was randomized to superficial closure of the K-L incision with a running subcuticular 3 0 monocryl suture then sealed with OCA skin adhesive.
Lovastatin production did not increase significantly with superficial air velocity in the 7 L reactor.
This indicated that a change in specific power input would change the k L a significantly than the change in the superficial gas velocity.
It relates k L a to the specific power input and the superficial gas velocity as given in the following: (9) k L a = δ (P g V L ) β v s α.
The k L a values obtained as a function of power input, superficial gas velocity, and palm oil fraction were validated by two different empirical equations.
In healthy cartilage, L-PGDS immunostaining was located in only a few cells in the superficial and middle zones.
l Orbicularis oris muscle was overlapped and sutured with three 5-0 nylon stitches in the manner of deep to superficial and superficial to deep.
The superficial head of the pronator quadratus (PQ) was released in an L-shaped fashion.
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