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Interaction with kinetic site 1 is characterized by relatively fast attachment and slow detachment, whereas attachment to and detachment from kinetic site 2 is fast.
Inactivation of viruses and interaction with kinetic site 2 provide only a minor contribution to removal.
Solute and AgNP breakthrough was quantified by applying numerical models considering one kinetic site model for particle transport.
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The two codes were modified by incorporating reversible adsorption to two types of kinetic sites.
Employing a two-site kinetic model to simulate breakthrough curves, attachment/detachment to two types of kinetic sites as well as inactivation of free and attached viruses were evaluated.
In contrast, model parameters describing the sorption capacity of the sand grains and the fraction of kinetic sorption sites on the sand decreased with increasing ionic strength.
In the presence of a lag phase that allows appreciable amounts of chemical to diffuse into kinetic sorption sites, nonequilibrium sorption will only impede degradation.
Kinetic and active site binding studies suggested mono-dentate binding of thiadiazole, oxadiazole, and triazole rings to the active site dicopper center of tyrosinase including hydrophobicity contributing to the potent inhibition.
Ostensibly higher step edge energy for defect-assisted nucleation at higher temperature appears to be compensated by an increase in the combined kinetic coefficient and site density for defect-assisted nucleation.
The model describes the structure and growth of planar PAH molecules, and is referred to as the kinetic Monte Carlo aromatic site (KMC-ARS) model.
Two models are employed for this study: a detailed PAH growth model, referred to as the kinetic Monte Carlo aromatic site (KMC-ARS) model [Raj et al., Combust. Flame 156 (2009) 896 913]; and a multivariate PAH population balance model, referred to as the PAH primary particle (PAH-PP) model.
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