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The standard binding free energy value is close to the experimental Kd in the nanomolar range.
A genetic predisposition has been suggested by higher rates of KD in the siblings as well as in the children of parents with a history of KD in childhood [1, 2, 3, 4, 5].
It has been suggested that early initiation of KD in PDH deficiency might improve patients' long-term outcome [7].
Our results are suggestive for the absence of efficacy of KD in CTTH patients, unlike in episodic migraineurs.
To evaluate the predictive efficacies of 4 existing scoring systems for intravenous immunoglobulin (IVIG) resistance in Kawasaki disease (KD) in hospitalized children with KD in a children's hospital affiliated with the Capital Institute of Pediatrics, Beijing, China.
To determine the incidence of Kawasaki disease (KD) in Northern France by using new American Heart Association AHAA) criteria.
Complexes with Kd in the nanomolar range clearly show stronger killing activity than weaker interactions [17].
Our data suggested that SUV39H1 KD in cancer cells have shorter telomeres.
In summary, we conducted a GWAS for KD in a Han Chinese population.
HLA-Bw22 (Bw54) has been associated with KD in a Japanese population [14], and a SNP located in the HLA-E gene was suggested to be associated with KD in the Han Chinese population [15].
A persistent infectious agent could also explain recurrences of KD in at least 1 3% of affected children [1].
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