Silvia Camporesi, an ethicist at King's College London, observed: "It is plausible to think that in 50 years, or maybe less, the 'natural' able-bodied athletes will just appear anachronistic".
Since an increased viral burden in smoke-exposed animals did not appear to be contributing to the exaggerated inflammation observed in those animals, it is plausible to argue that a smoke-exposed lung is responding differently to a viral stimulus, which may be contributing to the exaggerated response.
It is plausible that the observed in macromitophagy-deficient atg32Δ cells age-related changes in the membrane lipidomes of mitochondria, the ER and the PM may shorten yeast longevity by establishing a pro-aging cellular pattern.
Moreover, it is plausible that the observed in yeast grown in the presence of exogenous LCA build-up within the mitochondrial matrix of cristae disconnected from the IMM and thus exhibiting flat bilayer conformation was caused by the substantial rise in the relative levels of bilayer forming (i.e., cylinder-shaped) mitochondrial glycerophospholipids seen in these cells.
Thus, it is plausible that the redshift observed at higher pH is attributed to a relatively higher dipole moment of MUA as it is deprotonated.
It is plausible that diffuse opacities observed in this study might be attributed to high fluoride levels in the drinking water.
Since the adipose tissue, particularly from visceral fat depots, is capable of producing pro-inflammatory cytokines that induce hepatic production of CRP and other acute phase proteins, it is plausible that the association observed is mediated by obesity.
Therefore, it is plausible that the resistance to fluconazole that we observed with fet3Δ/fet3Δ and ftr1Δ/ftr1Δ mutants might be an influence on the drug's target, rather than its import.
First, it is plausible that the induced resistance observed in the laboratory trial does not hold during the course of the field experiment.
