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However, this may be a consequence of residual kinase activity on incomplete depletion or inactivation of Aurora B. As the ability of Aurora B to correct improper attachments may rely on increased distance from its kinetochore substrates, it is logical to ask whether its function in the SAC is regulated in the same manner.
So it is logical to ask whether the rhetoric is being exaggerated.
In view of the still unproven decisively intensive "statin as monotherapy" strategy and "residual risk" concept, it is logical to ask whether other strategies, particularly fibrate/statin combination therapy, could be more beneficial and safer.
However, given the broad patterns of co-expression and functional redundancy observed between co-members of various Sox families across the Sox phylogeny [ 27, 31, 32, 37- 49] 49], it is logical to ask whether such partial redundancy is a shared ancestral feature or the result of convergent evolution.
Alas, until now the bitterest comparator for the "intensive" statin monotherapy was 'usual-dose" statin monotherapy... then, in view of the lack of a proven effect of the current intensive "statin as monotherapy" strategy and "residual risk" concept, it is logical to ask whether other strategies could be more beneficial and safer [ 10].
Similar(55)
And they're also twins, so it was logical to ask them to take part.
It's logical to ask, so what if it was only four members?
It was logical to ask the question: were there changes in genes associated with these processes?
It is therefore logical to ask whether other genes involved in the alternative complement pathway may influence the risk.
It is completely logical to ask: why don't they just get one?
"It was really logical to ask them to pay for it".
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