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To address these issues, we characterized MGMT promoter methylation patterns using bisulfite sequencing to obtain quantitative methylation results for all 97 CpG sites in 70 newly diagnosed GBM patients.
To begin to address these issues, we characterized initial responses of tumor vessels to VEGF blockade in preclinical tumors with a range of responses to anti-VEGF therapy (sensitive, moderately responsive, and resistant).
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To shed light on this issue, we characterized cell death in primary OA chondrocytes mediated by the CD95 (Fas) pathway.
To investigate this issue, we characterized p65-dependent apoptosis using p65 null MEFs, which are resistant to genotoxin-induced apoptosis.
In order to address this issue, we characterized the gene expression profiles of A549 lung cancer cells and their mtDNA-depleted ρ0 counterparts grown in culture and as tumor xenografts in immune-deficient mice.
To overcome these issues, we first characterized the relationship between flow velocity and the region where Helmholtz resonance is generated.
To shed further light on these issues, we have characterized by protein engineering the structure of an expanded but native-like intermediate that accumulates transiently in the unfolding reaction of the small protein S6 in the presence of SDS.
To explore these issues we have characterized novel WSSV samples from five countries in Asia, revisited the ones already published, and generated a simple model of genome shrinkage.
To address abovementioned issues, we first characterized each drug-target pair from the views of both drugs and targets, respectively.
To address the issues described above, we characterized the DNA cleavage activity of the catalytic core of human topoisomerase IIα.
To address this issue, we have characterized the transcriptional landscape of radial glial precursors (RPs) in the embryonic murine cortex.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com