In addition, quiescence has been postulated to prevent stem cell exhaustion.
However, the RQ 135-1 sequencontainsins highly developed secondary structures that have been postulated to prevent complementary and strands of RNA from annealing to each other during replication leading to efficient and very rapid replication [ 19, 20].
Such infections have been postulated to prevent IgE mediated allergic diseases by blocking effector cell IgE receptors with parasite induced specific and polyclonal IgE or by production of the anti-inflammatory cytokine interleukin-10 [ 10].
This delay in acinar cell maturation has been postulated to prevent expression of cellular antigens at the critical time of self-tolerance, resulting in inefficient clonal deletion of acinar tissue-reactive T cells.
As quiescence has been postulated to prevent premature stem cell exhaustion during aging (Orford & Scadden, 2008), our observation of decreased frequency of quiescent LT-HSCs in Smurf2-deficient mice suggests that Smurf2-deficient HSCs may experience premature exhaustion.
The integrity of the trabeculodescemetic window allows controlled outflow of aqueous humor, which reduces the risk of profound and long-term hypotony, and it also has been postulated to prevent egress of cytokines and inflammatory mediators from the anterior chamber into the subconjunctival space, which reduces the risk of inflammation, scarring, and failure of filtering surgery [ 39– 41].
The cement-implant interface, which is postulated to act as a barrier to prevent osteolysis, was dominated by another mechanism, which cannot be explained by our detailed analysis.
Nogo-A, MAG, and OMgp activate RhoA through the NgR/p75 receptor complex, and this NgR/p75-complex/RhoA pathway is postulated to be responsible for the inhibitory signals that prevent axon regeneration [5].
