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TRALI is postulated to develop as the result of two separate clinical events [ 15, 16].
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Although KS-related chylothorax has been postulated to develop due to metastatic KS of the thoracic duct [ 16], our findings suggest that chylothorax may arise due to development of in-situ KS in this region.
The latter concept is analyzed in its general properties and is postulated to underpin the stability of the developing Bauplan down to the ultimate conserved details.
As IL-6 is postulated to play a pivotal role in the pathogenesis and disease activity of sJIA, MRA is being developed in this indication.
The relationship is postulated to follow the Mooney Rivlin function.
The growth is postulated to occur in two stages.
MFP is postulated to be typically self-limited whereas FM is postulated as chronic.
RARβ2 is postulated to be a tumor suppressor gene.
In cirrhosis, tissue hypoxia is postulated to stimulate angiogenesis [ 59].
BRCA1 protein is postulated to function as a tumour suppressor.
Although KS-related chylothorax formation was initially postulated to develop due to metastatic KS to the thoracic duct [ 118], more recent findings suggest that this may arise due to development of in-situ KS in this region [ 123].
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