Such associations are plausible through several biological mechanisms, including alterations to vasomotor tone [33], [34], thrombogenic effects [35] and the induction of systemic inflammation [36].
Some believe that it is plausible that, through adequate information, we could largely correct mistaken beliefs about the link between genetic and personal identity, and thus reduce the risk of problematic expectations toward the clone (Harris, 1997, 2004; Tooley 1998, 84 5; Brock, 1998, Pence, 1998).
As such, it is plausible that through modification of competitive interactions within and between niches rather than creation of novel niches, resident niche occupation could potentially promote or inhibit diversification of an invading lineage.
Thus, it is plausible that through de novo synthesis and deposition of collagen I, PSCs accompany and favour pancreatic cancer cell metastasis by providing trails of least resistance for cells to adhere and migrate.
Given the small size of the KIR2DL1-Cw6 complex, and the fact that signaling through KIR2DL1 is initiated by tyrosine phosphorylation of its cytoplasmic domain ITIM motif by Src kinases, it is plausible that KIR2DL1 signals through the kinetic-segregation mechanism.
While not directly addressed by this study, it is plausible that continuous stimulation through the T cell receptor in HIV infected individuals may induce chronic rather than transient FOXP3 upregulation, resulting in dysfunctional effector T cells.
The generally accepted mechanism of cell death is through hyperthermia, but it is plausible that destruction of the cell through mechanical means could also play a significant role.
In addition, it is plausible that tef is regulated through the circadian clock as well as a direct light pathway.
Since the intracellular concentration of denatured proteins impacts HSF1 activity through sequestration of HSPs [10], [36], [37], an indirect link between PRKCI and HSF1 activity through SQSTM1 is plausible.
