As every IxAC can inhibit the AP in all mammals tested, we conclude that diversification is not due to host specificity within the Mammalia class.
As incubation of the biomass after the addition of exogenous E1 before pretreatment did not have any discernible impact on enzymatic conversion, we conclude that the effect is not due to residual post-pretreatment E1 activity.
At this point it can be concluded that lag phase is not due to the physiology of yeast, but it may be due to the resistance in internal diffusion of glucose.
It is concluded that the deterioration is not due to oxidation of graphite to CO2 or CO but to the mechanical disintegration of the electrocatalyst structure as well as the formation of solid graphite oxides.
We concluded that spatial vision is not due to the presence of more (3 7 in our larva) photoreceptors in the eyes, since all photoreceptors from one eye synapse on the same primary interneuron.
Thus, we conclude that PL-induced autophagy is not due to disruption of cellular bioenergetics.
We conclude that the lack of phenotype is not due to a founder effect and/or to (minimal) transgene mis-expression.
Thus we conclude that the change in methionine concentration is not due to an increase in total protein concentration.
Therefore it can be concluded that the enhanced inhibitory effect is not due to additive action of two antimicrobial agents.
We conclude that the aromatase promoter transcript increases observed is not due to gonadotropin sensitivity.
