Exact(4)
The nature of the representations in these areas is, however, incompletely understood, and has only been studied using visual stimuli.
The molecular basis of the contribution of CenH3 to centromere/kinetochore functions is, however, incompletely understood, as its interactions with the rest of centromere/kinetochore components remain largely uncharacterised at the molecular/structural level.
What determines the switch between disease resolution and progression is however incompletely understood.
The role of COX2 in the systemic vasculature in humans with early type 1 diabetes is, however, incompletely understood (8– 11).
Similar(56)
The precise molecular mechanisms responsible for differentiation of circulating monocytes into tissue macrophages are, however, incompletely defined.
The upstream signal transduction mechanisms leading to increased NF-κB activity are, however, incompletely characterized.
Mechanisms that link metabolic syndrome to increased risk are, however, incompletely understood.
The structural basis for enterococcal serotypes, however, is still incompletely understood.
The molecular mechanism of the effect is however only incompletely understood [ 7, 8].
The molecular basis of transition to ADI is still incompletely characterized, however several androgen receptor-based hypotheses have been formulated [8], and they share the common denominator that AR acquires the ability to signal even in the androgen-depleted or AR-inhibited environment [9].
However, it is still incompletely understood whether isoflurane exerts its protective role through preventing mitochondrial permeability transition pore (MPTP) opening.
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