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Confirmation is by demonstration of the virus in samples sent to a laboratory.
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This is substantiated by demonstration of several of elevated N-acylethanolamine analogues related to AEA that are also metabolized by FAAH.
Cerebrospinal fluid (CSF) pleocytosis is considered a condition sine qua non for the diagnosis of CNS involvement in TBE, which in routine clinical practice is confirmed by demonstration of serum IgM and IgG antibodies to TBEV [ 2- 4].
Cerebrospinal fluid pleocytosis is considered a condition sine qua non for the diagnosis of neurologic involvement in tick borne encephalitis, which in routine clinical practice is confirmed by demonstration of serum IgM and IgG antibodies to tick borne encephalitis virus.
Diagnosis is confirmed by demonstration of the infarcted areas by computed tomography (CT) scanning.
Diagnosis is made by demonstration of the eggs in the host's faeces, biliary, or duodenal aspirates.
The diagnosis is made by demonstration of Entamoeba histolytica cysts or ameba in the pus or positive serology [ 2, 5].
Diagnosis of Cheyletiella infestation is made by demonstration of the mite or eggs in superficial skin scrapings, transparent tape preparations or by flea combing.
This is supported by demonstration of potentiated toxicity through a mechanism believed to involve the Ah receptor as a site of loss with respect to toxicity.
This is illustrated by demonstration of increased vitronectin in both chronic inflammatory liver disease and in liver tumours (Jaskiewicz et al, 1993; Nejjari et al, 2002).
The validity of these management changes was confirmed in a high proportion of cases and was supported by demonstration of superior prognostic stratification by PET compared with conventional staging in several series (Table 1).
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