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In liver, the enzyme is bound to cell surface proteoglycans within the sinusoids, from where it can be released by heparin.
Therefore it is likely that at least some of the mitogenicity in AEF is bound to cell surface heparin-related molecules and in this manner is presented to the oligodendrocyte surface as a result of cell cell contact.
In particular, WASP is bound to cell membranes; at some point it must detach from the Arp2/3 complex so that the nearness of the membrane does not interfere with the growth of branches.
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We hypothesize that serum IgE does not necessarily reflect total body IgE because in vivo IgE can be bound to cell surface receptors such as FcεRI and FcεRII (CD23).
However, most guidance factors in the nervous system are bound to cell membranes or the extracellular matrix.
We surmise that the rebound virus may result from the release of progeny virus that had before been bound to cell receptors because of the inhibition of viral neuraminidase activity by oseltamivir.
Measuring the amount of bound Gd3+ from γ-counting revealed that the Gd3+ ions were bound to cells at ∼0.1 fmole Gd/cell (P<0.01).
Individual sensor nodes in the network are bound to cells of the virtual hexagonal tessellation based on their geographic locations.
CHIKV particles were bound to cells pretreated with cytoskeleton depolymerizating agents.
Cells were prechilled to 4°C for 15 min, media were aspirated and ∼200 300 ng AF594 labeled virus was bound to cells for 1 h at 4°C.
Virions were bound to cells on ice for 1 h in the presence of drugs.
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