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The objective of this project is to investigate activation of a local renin-angiotensin system in the heart during myocardial ischemia and to assess the role of angiotensin in modulating adrenergic neurotransmission.
Participants completed a rapid serial visual presentation paradigm designed to investigate activation of attentional networks to behaviorally-relevant targets and contingent attention capture by task-irrelevant distractors.
To further investigate activation of the complement system by the different influenza virus infections, immunoblot analyses of BAL were performed.
To corroborate the findings that cocaine and gp120-mediated toxinvolvedvolved the apoptotic pathway, we next sought to investigate activation of caspase-3.
To investigate activation of c-MET in the drug-resistant cells, we confirmed the levels of p-MET protein.
To investigate activation of calpains in our model, we monitored the formation of α-spectrin breakdown products (SBDPs) by western blot.
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Several recent studies investigated activation of glycolysis during brain hypoxia exposure.
We investigated activation of checkpoint signaling and induction of cell cycle arrest following exposure of iPS cells to γ-radiation.
We next investigated activation of the Akt signaling pathway in response to G-CSF, which we have previously shown to be important in G-CSF-mediated cell survival and proliferation.
Preparation of metal-free enzymes is required for investigating activation of many metalloenzymes.
To find an explanation for this dissociation of phenotypes, we investigated activation of IGF signaling under acute oxidative stress.
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