As we present in the introduction, the expression dependences between sets of coexpressed genes, as well as between the processes these sets of coexpressed genes carry out, would never be linear.
The introduction of mesopores enhanced the expression of ALP and BSP in the cells grown on the mesoporous HA coatings, on the premise of maintaining the protein expression in a sequence to ensure the correct temporo-spatial expression in osteogenesis.
Consistent with the in vivo data, co-expression with Gαolf dramatically increased levels of AC5, and vice versa, the introduction of AC5 enhanced the expression of Gαolf.
However, the introduction of proofreading complicates the expression of and to such a point that no testable relationship between and can be derived so that the model can no longer be validated by experimental data.
As stated above in the introduction, controversial reports exist regarding the expression of LRP1b in human tissues ranging from restricted [4] to broad expression [2,5].
As mentioned in the introduction, the studies demonstrated that the expression of CXCL10 and CXCR3 was increased in the collagen-induced arthritis model, and neutralizing anti-CXCL10 antibodies ameliorated disease manifestation in these models [ 52, 53].
Notably, treatment of these LARP7 KD cells with flavopiridol or introduction of siCDK9s strongly inhibited the expression of four of those genes: Slug, FOXC2, ZEB2, and Twist1, suggesting that P-TEFb may directly affect their expression.
Because we have observed miR-29b-mediated reduction of migration ability and gene expression regulation, we asked whether miR-29b introduction has an impact on the expression of EMT-associated proteins.
In the case of Gata1, its introduction into GMPs results in the expression of erythroid potential with the appearance of large blast-like multipotential colonies in vitro (Heyworth et al., 2002).
