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Does it intrinsically lack the necessary mainstream-mobilising language that makes me run towards it?
Much of the literature that currently exists comes from administrative claims data, which intrinsically lack insight into why individuals stopped or never started a DMARD regimen.
These observations conflict with a recent study in SMs that reported that natural hosts intrinsically lack the ability to respond to TLR stimulation in vitro [ 18].
Because apoE−/− mice (C57BL/6 background) intrinsically lack sPLA2-IIA due to a natural mutation [ 56], the anti-atherosclerotic effect of A-002 could be attributable to the inhibition of other sPLA2 isoforms, probably sPLA2-V or –X (note that A-002 does not inhibit sPLA2-III, an atypical sPLA2).
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"People with disabilities," he recalled, "were considered intrinsically lacking; they were thought of as not whole, not even fully human".
The eight-part series chronicles a season in the life of the country's best cheerleading squad as it tries for a third year running to win the national championship — though the drama of the incumbent intrinsically lacks real excitement.
All moral entities, however, of whatever type, depend for their existence on the will of intelligent beings who externally assign normative, action-directing significance to things intrinsically lacking this, but nonetheless somehow compatible with or receptive to it.
The same study showed that an atypical EPEC strain, E128012, which intrinsically lacks EAF, is also virulent in volunteers.
A tagT deletion strain intrinsically lacks H1-T6SS acthereforeherefore, interpreting its inability to respond to the antibiotic as evidence of TPP involvement is problematic (Basler et al., 2013; Casabona et al., 2013; Ho et al., 2013).
The principal experimental evidence for the potential role of mammalian sPLA2 in atherosclerosis has arisen from studies employing transgenic mice overexpressing human sPLA2-IIA (PLA2G2A-Tg) [ 55], beyond the fact that the C57BL/6 mouse strain intrinsically lacks sPLA2-IIA as a result of a natural mutation of its gene [ 56].
Importantly, resting CD4+ T cells, in which SAMHD1 expression was (i) experimentally silenced by knockdown strategies or (ii) intrinsically lacking due to non-sense mutations in the samhd1 gene, supported the early phase of HIV-1 infection including the completion of RT and early gene expression.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com