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Good functional outcome was achieved significantly more often in patients with meningoencephalitis, epilepsy, GBS/MG, and intoxication (P < 0.01).
All four health/behavior variables interacted with baseline alcohol intoxication (p = 0.001), enforcing a stratification based on presence of early alcohol intoxication or not (Table 5).
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Nifedipine ingestion was associated with less severe intoxications (p < 0.0001; OR 0.08 [0.08–0.08).
This trend was not found for symptoms of anxiety where there were no associations with intoxication categories (p value for trend: 0.679) and alcohol consumption categories (p value for trend: 0.061).
CCl4 intoxication decreased (P < 0.05) the level of T3 and T4.
However, 50 mg/kg of silymarin and various doses of SAME augmented the CCl4-induced intoxication and significantly (P < 0.01) recovered the NORs per cell, body weight, liver weight, and relative liver weight.
Post-treatment of LME at 100 mg/kg b.w and 200 mg/kg b.w, dose improved the CCl4 intoxication and extensively reduced (p < 0.01) the lung weight, relative lung weight, % DNA damages and AgNORs count as compare to CCl4 group.
In univariate analysis, survival was lower for patient with refractory cardiac arrest unrelated to drug intoxication, 13 vs 50%, respectively, p = 0.03.
Cisplatin intoxication resulted in significant decrease (P < 0.05) in renal antioxidant enzyme activities like SOD, CAT, GPx, GR, and GST in CP-treated rats compared with the control group.
Metformin intoxication progressively inhibited global oxygen extraction (P = 0.008) and consumption (P < 0.001) and increased mixed venous oxygen saturation (from 65±± 6 up to 78 ± 7%, P < 0.001), despite no change in oxygen delivery (P = 0.58).
ILE reversed cataleptic scores, miotic pupils, and hypothermia of HA intoxication much faster than normal saline (P < 0.001).
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Justyna Jupowicz-Kozak
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